您好,S22668866!歡迎加入維基百科!
感謝您對維基百科的興趣與貢獻,希望您會喜歡這裏。除了歡迎辭以外,也請您了解以下重要文章:
![]() |
![]() |
![]() |
![]() If you have any questions about the Chinese Wikipedia, please leave a message here. Thank you for visiting!
|
- 閱讀新手應該注意的七種常見錯誤、理解維基百科的立場與常見注意事項。
- 有任何編輯上的問題?請試試IRC即時討論。
- 不知道有甚麼可寫?條目請求、最多語言版本的待撰條目、缺少的傳統百科全書條目和首頁的缺失條目中列出了許多維基百科目前還沒有的條目,歡迎您來撰寫!
- 希望您能享受共同編寫百科的樂趣,成為一名充實的維基百科人。..
-- 用心閣(對話頁) 2013年6月1日 (六) 12:03 (UTC)
==
==
此使用者目前已被封鎖。 以下為最近的封鎖紀錄以供參考:
2019年11月28日 (四) 18:21 Mys 721tx 討論 貢獻已封鎖S22668866 討論 貢獻期限為1 週(停用帳號建立) (屢次增加不實資料)
- 我認為這是一個嚴重的毀謗
- 關於毒品 和快樂中樞的相關資料非常多 繁衍會刺激快樂中樞的資料 也同時有說到
- http://www.epochtimes.com/b5/7/2/1/n1610828.htm
- https://blog.xuite.net/upvna/twblog/193966822-%E8%85%A6%E9%83%A8%E7%9A%84%E5%BF%AB%E6%A8%82%E4%B8%AD%E6%A8%9E
- http://enc.nutn.edu.tw/Page/Index/%E6%AF%92%E5%93%81%E5%88%86%E7%B4%9A%E4%BB%8B%E7%B4%B9
- https://antidrug.moj.gov.tw/lp-1211-2.html 甚至政府的官方網站也有
- 以及醫學院公開發表的醫學報告也有 https://ksph.kcg.gov.tw/walking04.htm
- https://www.therecoveryvillage.com/heroin-addiction/heroin-affects-nervous-system/#gref 海洛英影響大腦的關鍵區域與愉悅途徑有關,包括額葉皮層和伏隔核。
- 我覺得這種不實指控 非常扯
- 沒有經過考究 就指控我提供不實資料 毒品相關的論文各種考據各項論文都有
- 我在這裏申訴 這是一項嚴重的毀謗 毀謗我提供不實資料
- 況且 不要說哪裏的論文 連不同語言的維基百科也有提到 https://en.m.wikipedia.org/wiki/Reward_system
- 關於快樂中樞和毒品和繁衍的關聯性 我告訴你 你們怎不把每個條目都刪光光
- 這種行為等同於正在封鎖一個消息 沒有經過考究 就連不同語言的WIKI都有提到快樂中樞和毒品和繁衍的關聯性
- 我強烈譴責 這種封鎖消息的行為 且我要求 恢復我所發佈的資料
- 相關依據上面都有 就連不同語言的維基百科裏都有寫到 https://en.m.wikipedia.org/wiki/Reward_system 在WIKI中相關來源也都有 沒有任何理由可以說我提供不實資料 我非常強烈譴責這個指控
- 沒有經過考究 就說我提供不實資料 這就是毀謗
- 我現在證據我都附上了 這不實的指控 居然沒有解除
- 要求來源可以 但是不實的指控我提供不實資料
- 這就是毀謗 我強烈譴責這種行為
- 管理員沒有進行考究 就對使用者毀謗
- 這是非常惡劣的指控 已經侮辱了維基百科的使用者
- 我也拿出證據上訴了 居然還是指控我提供不實資料 這是不合理的
- 再者本來就要進行考究 才能扣別人帽子 現在就是對於我的毀謗
- 我非常強烈譴責這項行為
- 上面是被封鎖一周
- 以下是被封鎖一個月的申訴
- 請還我一個公道謝謝
- (:)回應:請閣下詳細說明該來源中哪段內容有提到你所增加的內容。謝謝。--SCP-2000 2019年12月10日 (二) 20:16 (UTC)
- (!)意見 申訴者只是第二次因違反方針而被封禁,且申訴者只是在單一條目內進行違反方針之行為,再者其他人及該名管理員未有向他發出警告,未能讓他完全得知其情況,故私認為沒有封一個月的必要及應予縮減封禁時間。--SCP-2000 2019年12月10日 (二) 20:16 (UTC)
- "這是一項沒有理由的刪除 相關毒品的論文 國內外都有 我寫的這段就是毒品的真相"、 "已閱讀內容"。具備檢查頁面歷史的能力應當是討論封禁必要性資格的必要條件。-Mys_721tx(留言) 2019年12月10日 (二) 20:38 (UTC)
- 謝謝閣下意見,基本上每次討論我會先看當事人的貢獻及頁面歷史。另外,私不認為未經警告下,就直接進行封禁是合理及符合封禁方針的。謝謝。--SCP-2000 2019年12月10日 (二) 21:34 (UTC)
- 翻了一下條目歷史,有個層級2級的警告。Itcfangye(留言) 2019年12月11日 (三) 05:36 (UTC)
- (!)意見 申訴者只是第二次因違反方針而被封禁,且申訴者只是在單一條目內進行違反方針之行為,再者其他人及該名管理員未有向他發出警告,未能讓他完全得知其情況,故私認為沒有封一個月的必要及應予縮減封禁時間。--SCP-2000 2019年12月10日 (二) 20:16 (UTC)
- 我使用的來源
- https://www.therecoveryvillage.com/heroin-addiction/heroin-affects-nervous-system/#gref
- 來源中這段有說到我所增加的內容
- The key areas of the brain that are impacted by heroin are related to the reward or pleasure pathways and they include the frontal cortex and the nucleus accumbens. These circuits are important for natural rewards such as what comes when you enjoy a meal or have sex.--WW(留言)
- 我想補充一點,其實在毒品的不同語言維基百科中,都有十分可靠的來源,顯現了我所增加的內容,舉例來說,
- https://en.m.wikipedia.org/wiki/Reward_system ,這裏面有提到,
- The brain structures that compose the reward system are located primarily within the cortico-basal ganglia-thalamo-cortical loop,
- The reward system is a group of neural structures responsible for incentive salience (ie, motivation and "wanting"
- particularly ones which involve pleasure as a core component
- 有提到大腦中樞包含着獎勵系統,負責刺激慾望,並且以愉悅感為核心
- Primary rewards are a class of rewarding stimuli which facilitate the survival of one's self and offspring , and include homeostatic (eg, palatable food ) and reproductive (eg, sexual contact and parental investment ) rewards.[1]
- 主要獎勵是一類獎勵性刺激,可促進一個人的自身和後代的生存 ,並包括體內平衡(例如, 美味食品 )和生殖(例如, 性行為和親代投資 )獎勵。
- The hedonic hotspots located in the anterior OFC and posterior insula have been demonstrated to respond to orexin and opioids, as has the overlapping hedonic coldspot in the anterior insula and posterior OFC.[29] On the other hand, the parabrachial nucleus hotspot has only been demonstrated to respond to benzodiazepine receptor agonists.
- 已證明位於OFC前部和後島的享樂性熱點對食慾素和鴉片類藥物有反應,在前島區和後部OFC中有重疊的享樂性熱點。[2]
- 上述都有可靠的來源且,在條目所增加的內容,我是依據其他語言的毒品維基百科內容過來的,
- 如果認為我增加不實資料,我認為應該要提出可靠的依據來,且對其他語言WIKI也做出反應才對
- 然而也要有足夠的依據反駁上述來源--WW(留言)
參考資料
- ^ Schultz W (2015). "Neuronal reward and decision signals: from theories to data". Physiological Reviews. 95 (3): 853–951. doi:10.1152/physrev.00023.2014. PMC 4491543. PMID 26109341. "Rewards in operant conditioning are positive reinforcers. ... Operant behavior gives a good definition for rewards. Anything that makes an individual come back for more is a positive reinforcer and therefore a reward. Although it provides a good definition, positive reinforcement is only one of several reward functions. ... Rewards are attractive. They are motivating and make us exert an effort. ... Rewards induce approach behavior, also called appetitive or preparatory behavior, sexual behavior, and consummatory behavior. ... Thus any stimulus, object, event, activity, or situation that has the potential to make us approach and consume it is by definition a reward. ... Rewarding stimuli, objects, events, situations, and activities consist of several major components. First, rewards have basic sensory components (visual, auditory, somatosensory, gustatory, and olfactory) ... Second, rewards are salient and thus elicit attention, which are manifested as orienting responses (FIGURE 1, middle). The salience of rewards derives from three principal factors, namely, their physical intensity and impact (physical salience), their novelty and surprise (novelty/surprise salience), and their general motivational impact shared with punishers (motivational salience). A separate form not included in this scheme, incentive salience, primarily addresses dopamine function in addiction and refers only to approach behavior (as opposed to learning) ... Third, rewards have a value component that determines the positively motivating effects of rewards and is not contained in, nor explained by, the sensory and attentional components (FIGURE 1, right). This component reflects behavioral preferences and thus is subjective and only partially determined by physical parameters. Only this component constitutes what we understand as a reward. It mediates the specific behavioral reinforcing, approach generating, and emotional effects of rewards that are crucial for the organism’s survival and reproduction, whereas all other components are only supportive of these functions. ... Rewards can also be intrinsic to behavior (31, 546, 547). They contrast with extrinsic rewards that provide motivation for behavior and constitute the essence of operant behavior in laboratory tests. Intrinsic rewards are activities that are pleasurable on their own and are undertaken for their own sake, without being the means for getting extrinsic rewards. ... Intrinsic rewards are genuine rewards in their own right, as they induce learning, approach, and pleasure, like perfectioning, playing, and enjoying the piano. Although they can serve to condition higher order rewards, they are not conditioned, higher order rewards, as attaining their reward properties does not require pairing with an unconditioned reward. ... These emotions are also called liking (for pleasure) and wanting (for desire) in addiction research (471) and strongly support the learning and approach generating functions of reward."
- ^ Yager LM, Garcia AF, Wunsch AM, Ferguson SM (August 2015). "The ins and outs of the striatum: Role in drug addiction". Neuroscience. 301: 529–541. doi:10.1016/j.neuroscience.2015.06.033. PMC 4523218. PMID 26116518. "[The striatum] receives dopaminergic inputs from the ventral tegmental area (VTA) and the substantia nigra (SNr) and glutamatergic inputs from several areas, including the cortex, hippocampus, amygdala, and thalamus (Swanson, 1982; Phillipson and Griffiths, 1985; Finch, 1996; Groenewegen et al., 1999; Britt et al., 2012). These glutamatergic inputs make contact on the heads of dendritic spines of the striatal GABAergic medium spiny projection neurons (MSNs) whereas dopaminergic inputs synapse onto the spine neck, allowing for an important and complex interaction between these two inputs in modulation of MSN activity ... It should also be noted that there is a small population of neurons in the [nucleus accumbens] NAc that coexpress both D1 and D2 receptors, though this is largely restricted to the NAc shell (Bertran- Gonzalez et al., 2008). ... Neurons in the NAc core and NAc shell subdivisions also differ functionally. The NAc core is involved in the processing of conditioned stimuli whereas the NAc shell is more important in the processing of unconditioned stimuli; Classically, these two striatal MSN populations are thought to have opposing effects on basal ganglia output. Activation of the dMSNs causes a net excitation of the thalamus resulting in a positive cortical feedback loop; thereby acting as a 'go』 signal to initiate behavior. Activation of the iMSNs, however, causes a net inhibition of thalamic activity resulting in a negative cortical feedback loop and therefore serves as a 'brake’ to inhibit behavior ... there is also mounting evidence that iMSNs play a role in motivation and addiction (Lobo and Nestler, 2011; Grueter et al., 2013). For example, optogenetic activation of NAc core and shell iMSNs suppressed the development of a cocaine CPP whereas selective ablation of NAc core and shell iMSNs ... enhanced the development and the persistence of an amphetamine CPP (Durieux et al., 2009; Lobo et al., 2010). These findings suggest that iMSNs can bidirectionally modulate drug reward. ... Together these data suggest that iMSNs normally act to restrain drug-taking behavior and recruitment of these neurons may in fact be protective against the development of compulsive drug use."
希望您在編輯時,注意增添內容需要有可靠文獻支持您的論點。-千村狐兔(留言) 2019年12月23日 (一) 13:53 (UTC)
- 您是否有繞過封禁的行為?-千村狐兔(留言) 2019年12月29日 (日) 11:46 (UTC)
- @Mys 721tx: 請問S22668866君的傀儡在哪個條目進行編輯? 謝謝。--SCP-2000 按此申訴本人判斷 2019年12月29日 (日) 11:59 (UTC)
- Special:Contributions/1.65.160.18。-Mys_721tx(留言) 2019年12月30日 (一) 02:13 (UTC)
不相關,S22668866君是一次過增加大量原創研究內容,且以段落之方式撰寫。然而ip君是一次過增加較少原創研究內容,及以點列方式撰寫。再者S22668866君未曾編輯過有關香港及歷史的條目。--SCP-2000 按此申訴本人判斷 2019年12月30日 (一) 02:30 (UTC)
- ul/dl/ol在wikitext上僅一字符之差。分段及行文方式亦與本頁面文字相似。-Mys_721tx(留言) 2019年12月30日 (一) 05:08 (UTC)
- @Mys 721tx: S22668866君在這裏的行文方式為一行中有幾句句子,而ip君的行文方式為一行中有幾個詞語或短句。再者正常編輯時,大多會使用「:#*」這些符號,而非使用ul/dl/ol。--SCP-2000 按此申訴本人判斷 2019年12月30日 (一) 05:22 (UTC)
- Special:Contributions/1.65.160.18這些並不是我的編輯,而且1.65.160.18是香港IP,跟我完全不同WW(留言)
看起來是- ul/dl/ol在wikitext上僅一字符之差。分段及行文方式亦與本頁面文字相似。-Mys_721tx(留言) 2019年12月30日 (一) 05:08 (UTC)
- Special:Contributions/1.65.160.18。-Mys_721tx(留言) 2019年12月30日 (一) 02:13 (UTC)
- 知悉其解釋。應予新手善意推定。-千村狐兔(留言) 2020年1月3日 (五) 14:54 (UTC)
我沒有增加不實資料,上述申訴於1/12就申訴了,為什麼管理員沒有回覆? WW(留言) 2020年2月3日 (一) 02:25 (UTC)
- 吸毒比人類交配時更愉悅[來源請求],多巴胺增多意味着快感增多?--john doe 120@霍格華茲膜法學院時間魔法 2020年2月3日 (一) 03:44 (UTC)
- ^ Berridge KC (April 2012). "From prediction error to incentive salience: mesolimbic computation of reward motivation". Eur. J. Neurosci. 35 (7): 1124–1143. doi:10.1111/j.1460-9568.2012.07990.x. PMC 3325516. PMID 22487042. "Here I discuss how mesocorticolimbic mechanisms generate the motivation component of incentive salience. Incentive salience takes Pavlovian learning and memory as one input and as an equally important input takes neurobiological state factors (e.g. drug states, appetite states, satiety states) that can vary independently of learning. Neurobiological state changes can produce unlearned fluctuations or even reversals in the ability of a previously learned reward cue to trigger motivation. Such fluctuations in cue-triggered motivation can dramatically depart from all previously learned values about the associated reward outcome. ... Associative learning and prediction are important contributors to motivation for rewards. Learning gives incentive value to arbitrary cues such as a Pavlovian conditioned stimulus (CS) that is associated with a reward (unconditioned stimulus or UCS). Learned cues for reward are often potent triggers of desires. For example, learned cues can trigger normal appetites in everyone, and can sometimes trigger compulsive urges and relapse in addicts. Cue-triggered 'wanting』 for the UCS A brief CS encounter (or brief UCS encounter) often primes a pulse of elevated motivation to obtain and consume more reward UCS. This is a signature feature of incentive salience. Cue as attractive motivational magnets When a Pavlovian CS+ is attributed with incentive salience it not only triggers 'wanting』 for its UCS, but often the cue itself becomes highly attractive – even to an irrational degree. This cue attraction is another signature feature of incentive salience ... Two recognizable features of incentive salience are often visible that can be used in neuroscience experiments: (i) UCS-directed 'wanting』 – CS-triggered pulses of intensified 'wanting』 for the UCS reward; and (ii) CS-directed 'wanting』 – motivated attraction to the Pavlovian cue, which makes the arbitrary CS stimulus into a motivational magnet."